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primary antibody against sterol regulatory element binding protein 1c srebp1c  (Bioss)


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    Bioss primary antibody against sterol regulatory element binding protein 1c srebp1c
    Primary Antibody Against Sterol Regulatory Element Binding Protein 1c Srebp1c, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 51 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/primary antibody against sterol regulatory element binding protein 1c srebp1c/product/Bioss
    Average 94 stars, based on 51 article reviews
    primary antibody against sterol regulatory element binding protein 1c srebp1c - by Bioz Stars, 2026-02
    94/100 stars

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    Brusatol alleviates lipid dysregulation in IMQ-induced psoriatic mice by targeting IL-1β and subsequently activating the AMPK signaling pathway. A Representative western blots and B quantitative analysis of key proteins in the AMPK pathway in dorsal skin tissues from control (Con), model (Mod), and Brusatol-treated (Bru, 4.6 mg/kg) mice. Protein levels of total AMPKα, phosphorylated AMPKα (p-AMPKα), and downstream effectors for fatty acid oxidation (PPARα, CPT1A) and lipogenesis <t>(SREBP-1,</t> FASN, ACC1) were assessed, with β-actin as a loading control. C mRNA expression levels of <t>Srebp-1c</t> , Acc1 , Fasn , Pparα , and Cpt1a in dorsal skin tissues, as determined by RT-qPCR. Data in B and C are presented as mean ± SEM (n = 9 for ( B ); n = 5 for ( C )). * p < 0.05, ** p < 0.01, *** p < 0.001 vs . Mod group. # p <0.05 vs. control group.
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    Brusatol alleviates lipid dysregulation in IMQ-induced psoriatic mice by targeting IL-1β and subsequently activating the AMPK signaling pathway. A Representative western blots and B quantitative analysis of key proteins in the AMPK pathway in dorsal skin tissues from control (Con), model (Mod), and Brusatol-treated (Bru, 4.6 mg/kg) mice. Protein levels of total AMPKα, phosphorylated AMPKα (p-AMPKα), and downstream effectors for fatty acid oxidation (PPARα, CPT1A) and lipogenesis <t>(SREBP-1,</t> FASN, ACC1) were assessed, with β-actin as a loading control. C mRNA expression levels of <t>Srebp-1c</t> , Acc1 , Fasn , Pparα , and Cpt1a in dorsal skin tissues, as determined by RT-qPCR. Data in B and C are presented as mean ± SEM (n = 9 for ( B ); n = 5 for ( C )). * p < 0.05, ** p < 0.01, *** p < 0.001 vs . Mod group. # p <0.05 vs. control group.
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    Brusatol alleviates lipid dysregulation in IMQ-induced psoriatic mice by targeting IL-1β and subsequently activating the AMPK signaling pathway. A Representative western blots and B quantitative analysis of key proteins in the AMPK pathway in dorsal skin tissues from control (Con), model (Mod), and Brusatol-treated (Bru, 4.6 mg/kg) mice. Protein levels of total AMPKα, phosphorylated AMPKα (p-AMPKα), and downstream effectors for fatty acid oxidation (PPARα, CPT1A) and lipogenesis <t>(SREBP-1,</t> FASN, ACC1) were assessed, with β-actin as a loading control. C mRNA expression levels of <t>Srebp-1c</t> , Acc1 , Fasn , Pparα , and Cpt1a in dorsal skin tissues, as determined by RT-qPCR. Data in B and C are presented as mean ± SEM (n = 9 for ( B ); n = 5 for ( C )). * p < 0.05, ** p < 0.01, *** p < 0.001 vs . Mod group. # p <0.05 vs. control group.
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    Brusatol alleviates lipid dysregulation in IMQ-induced psoriatic mice by targeting IL-1β and subsequently activating the AMPK signaling pathway. A Representative western blots and B quantitative analysis of key proteins in the AMPK pathway in dorsal skin tissues from control (Con), model (Mod), and Brusatol-treated (Bru, 4.6 mg/kg) mice. Protein levels of total AMPKα, phosphorylated AMPKα (p-AMPKα), and downstream effectors for fatty acid oxidation (PPARα, CPT1A) and lipogenesis <t>(SREBP-1,</t> FASN, ACC1) were assessed, with β-actin as a loading control. C mRNA expression levels of <t>Srebp-1c</t> , Acc1 , Fasn , Pparα , and Cpt1a in dorsal skin tissues, as determined by RT-qPCR. Data in B and C are presented as mean ± SEM (n = 9 for ( B ); n = 5 for ( C )). * p < 0.05, ** p < 0.01, *** p < 0.001 vs . Mod group. # p <0.05 vs. control group.
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    Brusatol alleviates lipid dysregulation in IMQ-induced psoriatic mice by targeting IL-1β and subsequently activating the AMPK signaling pathway. A Representative western blots and B quantitative analysis of key proteins in the AMPK pathway in dorsal skin tissues from control (Con), model (Mod), and Brusatol-treated (Bru, 4.6 mg/kg) mice. Protein levels of total AMPKα, phosphorylated AMPKα (p-AMPKα), and downstream effectors for fatty acid oxidation (PPARα, CPT1A) and lipogenesis (SREBP-1, FASN, ACC1) were assessed, with β-actin as a loading control. C mRNA expression levels of Srebp-1c , Acc1 , Fasn , Pparα , and Cpt1a in dorsal skin tissues, as determined by RT-qPCR. Data in B and C are presented as mean ± SEM (n = 9 for ( B ); n = 5 for ( C )). * p < 0.05, ** p < 0.01, *** p < 0.001 vs . Mod group. # p <0.05 vs. control group.

    Journal: Chinese Medicine

    Article Title: Brusatol ameliorates psoriatic dyslipidemia by targeting IL-1β to restore AMPK-mediated lipid homeostasis

    doi: 10.1186/s13020-025-01287-8

    Figure Lengend Snippet: Brusatol alleviates lipid dysregulation in IMQ-induced psoriatic mice by targeting IL-1β and subsequently activating the AMPK signaling pathway. A Representative western blots and B quantitative analysis of key proteins in the AMPK pathway in dorsal skin tissues from control (Con), model (Mod), and Brusatol-treated (Bru, 4.6 mg/kg) mice. Protein levels of total AMPKα, phosphorylated AMPKα (p-AMPKα), and downstream effectors for fatty acid oxidation (PPARα, CPT1A) and lipogenesis (SREBP-1, FASN, ACC1) were assessed, with β-actin as a loading control. C mRNA expression levels of Srebp-1c , Acc1 , Fasn , Pparα , and Cpt1a in dorsal skin tissues, as determined by RT-qPCR. Data in B and C are presented as mean ± SEM (n = 9 for ( B ); n = 5 for ( C )). * p < 0.05, ** p < 0.01, *** p < 0.001 vs . Mod group. # p <0.05 vs. control group.

    Article Snippet: The primary antibodies including AMPKα (Rabbit, 66536–1-lg, Proteintech), p-AMPKα (Rabbit, 2535,CST), SREBP-1c (Rabbit, AF6288, Affinity), ACC1 (Rabbit, A19495, ABclonal), PPARα (Rabbit, 15540–1-AP, Proteintech), IL-1β (Rabbit, 16806–1-AP, Proteintech) and K17 (Rabbit, 13074, CST) were employed at a standardized dilution of 1:1000.

    Techniques: Western Blot, Control, Expressing, Quantitative RT-PCR